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Data interpretation is difficult because the observed modifications in hyperhomocysteinemic subjects may merely result from the occurrence of cardiovascular disorders
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The phosphorylated form of NAD + , NADP/NADPH, parallels NAD + /NADH by functioning as a redox coenzyme in the pentose phosphate pathway, generation of acetyl coenzyme A (acetyl-CoA) for lipid and cholesterol biosynthesis, and in the glutathione and thioredoxin antioxidant systems 4,5,8,9
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