glutathione scavenger in portuguese Inhibition of erythrocyte's catalase, peroxidase or peroxiredoxin 2 – Impact on cytosol and membrane Inflammatory bowel diseases: pathological

Inflammatory bowel diseases: pathological mechanisms and therapeutic perspectives | Molecular Biomedicine | Springer Nature Link

Binding of LL-37 to model biomembranes: Insight into target vs host cell recognition

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Autophagic liberation of endogenous TOR-activating substrates, such as glucose and glutamine, may therefore re-activate TOR to force mitotic progression through M phase and subsequently G1 arrest of the appressorium nucleus to limit continued rounds of mitosis, which is known to further require an S phase checkpoint to enable appressorium re-polarization [62]

Also shown to reduce the level of cytoplasmic and nuclear p53